Bioanalytical Project Manager and Manager 1

• Jun 2018 - Present

Medical Technologist

• Aug 2016 - Jul 2018

I worked in the Special Chemistry lab of the UNC McLendon Labs. I performed tasks associated with diagnostic testing of Serum Free Light Chains, Serum Protein Electrophoresis, Therapeutic Drug Monotoring, Drugs of Abuse Confirmation, and Carnitine/Acylcarnatines. I worked in the Special Chemistry lab of the UNC McLendon Labs. I performed tasks associated with diagnostic testing of Serum Free Light Chains, Serum Protein Electrophoresis, Therapeutic Drug Monotoring, Drugs of Abuse Confirmation, and Carnitine/Acylcarnatines.

Director of Research and Technology Development, Reagents and Assays

• Nov 2014 - Jun 2018

Project: Assay development for automated hematology/flow cytometry analyzer. • Optimize immunoassays for diagnostics. • Optimize buffer and assay conditions for hematological diagnostics. Project: Assay development for automated hematology/flow cytometry analyzer. • Optimize immunoassays for diagnostics. • Optimize buffer and assay conditions for hematological diagnostics.

Postdoctoral Associate

• Sep 2012 - Jun 2014

The Sue Jinks-Robertson Lab, has shown that specific types of mutations occur more frequently during high transcription in yeast. I designed a system in mammalian cells to better understand the processes that can lead to mutations and subsequently disease. This required the production of monoclonal cell lines that contain a single copy of a transgenic reporter that will express EGFP when a mutation occurs. The Sue Jinks-Robertson Lab, has shown that specific types of mutations occur more frequently during high transcription in yeast. I designed a system in mammalian cells to better understand the processes that can lead to mutations and subsequently disease. This required the production of monoclonal cell lines that contain a single copy of a transgenic reporter that will express EGFP when a mutation occurs.

PhD Candidate

• Aug 2006 - Aug 2012

My graduate lab studied the process of cellular immortalization with a focus on telomere homeostasis. Telomeres are structures on the ends of our chromosomes that protect our DNA similarly to how the plastic caps protect our shoe laces from fraying. My thesis work focused on a DNA damage response protein, hSnm1B, which localizes to telomeres. Snm1B, a required protein, is responsible for protecting telomeric DNA during replication and for repairing DNA from interstrand crosslinks (ICLs). ICLs are a very dangerous form of DNA damage that causes a covalent bond between the strands of DNA which blocks DNA replication. Many ICL inducing agents, such as cisplatin, are used in the clinic for treatment of cancer, but how these lesions are repaired by the DNA damage repair machinery of the cell is not fully understood. My work was the first to show an interaction between hSnm1B and PSF2, a protein required for replication and the repair of ICLs. Show less

Teaching Assistant Bio 49

• Sep 2010 - Dec 2010

Teaching Assistant for Bio 49S a freshman seminar course taught by Dr. James Reynolds. Teaching Assistant for Bio 49S a freshman seminar course taught by Dr. James Reynolds.

Research Technician

• Jul 2005 - Aug 2006

I worked with Dr. Mark Jutila on a high-throughput screen of antagonists of gamma delta T-cells. The screen used 96 well plates and flow cytometry to score the compounds. I ran the flow cytometer and organized the data on the screens. In addition I would retest positive hits to further characterize their activity. I worked with Dr. Mark Jutila on a high-throughput screen of antagonists of gamma delta T-cells. The screen used 96 well plates and flow cytometry to score the compounds. I ran the flow cytometer and organized the data on the screens. In addition I would retest positive hits to further characterize their activity.