Eugenia Prescott, Ph.D.

Senior Science Writer at IQ Solutions
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Contact Information
us****@****om
(386) 825-5501
Location
Columbia, Maryland, United States, US
Languages
  • English -

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Experience

    • United States
    • Public Relations and Communications Services
    • 100 - 200 Employee
    • Senior Science Writer
      • Jan 2023 - Present

    • United States
    • Hospitals and Health Care
    • 700 & Above Employee
    • Postdoctoral Research Fellow
      • Oct 2013 - Nov 2015

      Our lab is interested in CHAC1, a pro-apoptotic component of the PERK-ATF4-CHOP branch of the unfolded protein response (UPR) pathway, and its role in the development of atherosclerosis and heart disease. We originally identified CHAC1 using a systems biology study of inflammatory responses within a population of primary human aortic endothelial cells (HAEC) treated with the oxidized phospholipid ox-PAPC (Gargalovic, 2006). My project focuses on investigating the role of Chac1 in… Show more Our lab is interested in CHAC1, a pro-apoptotic component of the PERK-ATF4-CHOP branch of the unfolded protein response (UPR) pathway, and its role in the development of atherosclerosis and heart disease. We originally identified CHAC1 using a systems biology study of inflammatory responses within a population of primary human aortic endothelial cells (HAEC) treated with the oxidized phospholipid ox-PAPC (Gargalovic, 2006). My project focuses on investigating the role of Chac1 in cholesterol-induced macrophage apoptosis. I am interested in defining whether Chac1 participates in cholesterol-induced macrophage apoptosis, and pursuing future in vivo experiments in the well-established ApoE knockout (KO) atherosclerosis mouse model that may identify Chac1 as a therapeutic target. Show less Our lab is interested in CHAC1, a pro-apoptotic component of the PERK-ATF4-CHOP branch of the unfolded protein response (UPR) pathway, and its role in the development of atherosclerosis and heart disease. We originally identified CHAC1 using a systems biology study of inflammatory responses within a population of primary human aortic endothelial cells (HAEC) treated with the oxidized phospholipid ox-PAPC (Gargalovic, 2006). My project focuses on investigating the role of Chac1 in… Show more Our lab is interested in CHAC1, a pro-apoptotic component of the PERK-ATF4-CHOP branch of the unfolded protein response (UPR) pathway, and its role in the development of atherosclerosis and heart disease. We originally identified CHAC1 using a systems biology study of inflammatory responses within a population of primary human aortic endothelial cells (HAEC) treated with the oxidized phospholipid ox-PAPC (Gargalovic, 2006). My project focuses on investigating the role of Chac1 in cholesterol-induced macrophage apoptosis. I am interested in defining whether Chac1 participates in cholesterol-induced macrophage apoptosis, and pursuing future in vivo experiments in the well-established ApoE knockout (KO) atherosclerosis mouse model that may identify Chac1 as a therapeutic target. Show less

    • United States
    • Writing and Editing
    • 1 - 100 Employee
    • Scientific Writing Editor
      • May 2012 - Mar 2014

    • United States
    • Higher Education
    • 700 & Above Employee
    • Biochemistry Instructor
      • Jun 2013 - Jul 2013

      Summer Medical and Dental Education Program (SMDEP) Summer Medical and Dental Education Program (SMDEP)

    • United States
    • Higher Education
    • 700 & Above Employee
    • Private Tutor
      • Aug 2007 - Jul 2011

    • Doctoral Candidate
      • Aug 2005 - Mar 2011

      Designed, planned and successfully executed biochemical and genetic experiments focused on understanding the contribution of histone H3 and H4 proteins to gene regulation in the yeast Saccharomyces cerevisiae. Completed a Certificate in Teaching College Biology and the Preparing Future Faculty program.

Education

  • Duke University
    Doctor of Philosophy (Ph.D.), Biochemistry
    2005 - 2011
  • University of Northern Iowa
    Bachelor’s Degree, Chemistry
    2001 - 2005

Community

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